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diagnosis of invasive Candidiasis inNeutropenic children with cancer

Diagnosis of Invasive Candidiasis in Neutropenic Children with Cancer

by Determination of D-Arabinitol/L-Arabinitol Ratios in Urine

BERTIL CHRISTENSSON,1* THOMAS WIEBE,2 CHRISTINA PEHRSON,3

AND LENNART LARSSON3

Departments of Infectious Diseases,1 Pediatrics,2 and Medical Microbiology,3




Lund University Hospital, S-221 85, Lund, Sweden


Received 26 August 1996/Returned for modification 29 October 1996/Accepted 3 December 1996

Determination of D-arabinitol/L-arabinitol ratios (referred to as D/L-arabinitol ratios) in urine as a tool for



the diagnosis of invasive candidiasis was investigated in a prospective study comprising 100 children with

cancer. The analyses were made by gas chromatography. Positive D/L-arabinitol ratios were found for 10 of 10



children with confirmed invasive candidiasis, 12 of 23 patients undergoing empiric antifungal chemotherapy,

and 4 of 67 children not receiving antifungal treatment. D/L-Arabinitol ratios were positive 3 to 31 days



(median, 12 days) before the first culture-positive blood sample was drawn or empiric therapy was initiated.

The regular monitoring of D/L-arabinitol ratios in urine holds great promise as a sensitive method for



diagnosing invasive candidiasis in immunocompromised children with cancer. Moreover, it may be possible to

use an early rise in D/L-arabinitol ratios as a basis for the institution of antifungal chemotherapy and as a



means of avoiding unnecessary treatment with potentially toxic antifungal agents.


Invasive candidiasis is becoming increasingly common due

to the growing number of immunocompromised hosts. An

early and accurate diagnosis is of importance for improving

survival resulting from the institution of antifungal chemotherapy

(2, 15) and decreasing the unnecessary use of toxic antifungal

agents such as amphotericin B. Unfortunately, currently

available methods are not sufficiently sensitive and specific for

diagnosing invasive candidiasis (11, 19). Therefore, empiric

antifungal chemotherapy has been advocated for persistently

febrile granulocytopenic patients not responding to broadspectrum

antibiotic therapy (21). Although such empiric therapy

has been shown to decrease the frequency, morbidity, and

mortality of invasive fungal infection (21), inevitably, it also

leads to a potentially hazardous and extremely costly overtreatment

of some patients.

D-Arabinitol is a major metabolite of most Candida species,

and both D-arabinitol and L-arabinitol are present in normal



serum and urine. In an earlier study (9), we developed a gas

chromatographic method to determine the relative amounts of

D-arabinitol and L-arabinitol (referred to as the D/L-arabinitol

ratio) in urine. Elevated urine D/L-arabinitol ratios have previously



been found in a small number of patients with invasive

candidiasis (9, 16). Here, we report the results of a prospective

study of pediatric oncology patients designed to determine the

value of monitoring D/L-arabinitol ratios in urine for the early



diagnosis of invasive candidiasis.

MATERIALS AND METHODS


Patients. From March 1992 through October 1995, 100 children (age range, 1



to 17 years; mean age, 9 years) were prospectively studied at the Department of

Pediatrics, Division of Oncology, Lund University Hospital. All children were

considered to be at high risk for invasive candidiasis. Their malignant diagnoses

were acute leukemia for 47% of the patients, lymphomas for 13% of the patients,

and Wilms’ tumor for 14% of the patients; the remaining patients had various

solid tumors. All patients had central venous catheters and were receiving cytotoxic

chemotherapy. Broad-spectrum antibiotic therapy was instituted when patients

were febrile and granulocytopenic. All hospitalized patients were given

nystatin orally. During periods of hospitalization, the aim was to collect urine

samples at least twice weekly. The children who were too young to deliver urine

samples spontaneously and those who delivered only one sample were excluded

from the study. In all, 1,076 urine samples were collected.

A febrile (.38.38C), neutropenic (blood neutrophil counts, ,0.5 3 109/liter)



patient not responding to broad-spectrum antibiotic treatment was defined as

having invasive candidiasis when blood cultures were positive for Candida and/or

when Candida species were cultured or proven histopathologically in tissue



samples from normally sterile locations. Invasive candidiasis was clinically suspected

when a febrile, neutropenic patient did not respond to antibiotic treatment,

and in such cases, antifungal chemotherapy was empirically instituted even

when blood cultures were negative for Candida. Patients were defined as having



bacteremia when they responded to antibiotic treatment and blood cultures were

positive for bacteria but negative for Candida. Patients were also evaluated

during nonneutropenic periods, when all blood cultures were negative for Candida



and bacteria and no antifungal treatment was being given.

For patients with confirmed or suspected candidiasis, blood for culture was

always drawn at the time of urine collection. Additional blood cultures were also

performed once or twice daily for many of these patients. On the other hand,

during nonneutropenic periods without clinical suspicion of fungal infection, the

twice-weekly urine sampling was not always accompanied by blood culturing.

For comparison, single urine samples from 56 healthy nonhospitalized children

(age range, 1 to 15 years; mean age, 5 years) were also studied.

Samples. The urine samples were divided into 1-ml aliquots and were then

stored at 2208C before analysis for D/L-arabinitol ratios (see below). A biphasic



blood culture system (Septi-Check; Roche Products, Ska¨rholmen, Sweden) was

used to culture bacteria and fungi. For detection of fungi, blood was cultured

aerobically for 5 days at 378C, followed by 9 days at 308C; the cultures were



inspected daily for turbidity and were also shaken twice daily. Tissue samples

were cultured on Sabouraud agar and on agar with 4% horse erythrocytes for 7

days at 308C. Typing of the Candida organisms to the species level was done by



testing for production of chlamydospores and fermentation of glucose, galactose,

saccharose, maltose, lactose, and trehalose on rice agar (10).

D/L-Arabinitol analysis. Urine samples were filtered (0.45-mm-pore-size cellulose

acetate filters; Schleicher & Schuell, Dassel, Germany), and a 5- to 15-ml



portion of the filtrate was dried under a stream of nitrogen. Trifluoroacetic

anhydride and hexane (200 ml each) were added, and the preparations were

heated at 808C for 10 min and again dried. Thereafter, hexane (200 to 500 ml)

was added, and a 1- to 3-ml aliquot was used to obtain D/L-arabinitol ratios by gas



chromatography. As described previously (9), the method has an interassay

coefficient of variation of 7.7%. The total time for handling and analysis of each

sample was less than 1 h.

Study design. Samples were coded and were then submitted to the laboratory



and analyzed in blind batches. The results were not available to the pediatricians

treating the patients to ensure that the initiation of antifungal therapy would be

based solely on clinical observations and the results of blood or tissue cultures

and histopathology. The following was recorded for each patient: cytotoxic and

radiological therapy, antimicrobial therapy and prophylaxis, microbiological results,

and neutropenic and febrile events. Student’s two-tailed t test was used to

make comparisons between the D/L-arabinitol ratios for different groups. The



study was approved by the Ethics Committee at the Medical Faculty, University

* Corresponding author. Phone: 46 46 17 10 00. Fax: 46 46 13 74 14. of Lund.



636

RESULTS


The D/L-arabinitol ratios (mean 6 standard deviation) for

the 56 healthy nonhospitalized children were 2.0 6 0.6, and for

the 95 children with cancer, the ratios were 2.5 6 0.7 (P ,



0.01) during nonneutropenic periods. The latter group was

used to define the upper cutoff limit (mean ratio 1 3 standard

deviations, corresponding to a value of 4.6), and values of .4.6



were considered positive.

Ten patients with long-standing fever and neutropenia were

diagnosed with invasive candidiasis on the basis of blood cultures,

tissue cultures, and/or histopathology, and all these patients

had positive peak D/L-arabinitol results (Table 1; Fig. 1,

group A). By using multiple blood cultures, C. albicans was

isolated from five patients, C. parapsilosis was isolated from

three patients, C. tropicalis was isolated from one patient, and

C. glabrata was isolated from one patient. For the last patient,

postmortem tissue cultures were also positive for C. albicans.



Urine samples were available from 7 of 10 patients before

blood cultures were positive and showed increased D/L-arabinitol



ratios 3, 3, 4, 8, 9, 16, and 21 days, respectively, before the

first culture-positive blood sample was drawn. The time course

of the D/L-arabinitol ratios in one of these patients is presented



in Fig. 2. Five patients died from invasive candidiasis, and in

four of these patients the D/L-arabinitol ratio was positive 3, 8,



9, and 21 days, respectively, before the first culture-positive

blood sample was drawn (Table 1).

Antifungal chemotherapy (amphotericin B or fluconazole)

was instituted empirically (due to a lack of response after 3 to

5 days of broad-spectrum antibiotic treatment) for 23 neutropenic

and febrile patients with blood cultures negative for

Candida and bacteria. Twelve children had positive peak D/Larabinitol



ratio results (Table 2; Fig. 1, group B). Ten of 12

TABLE 1. Peak D/L-arabinitol ratios for febrile, neutropenic children with cancer and confirmed invasive candidiasis



Patient no. Primary disease Species

Peak D/Larabinitol



ratio

Time interval (days)

between positive

D/L-arabinitol



ratio and positive

blood culture

No. of positive

samples/no. of

all samplesa



Diagnosis based on: Patient

status

1 ALLb C. albicans 30 NAc 3/24 3 blood cultures Dead

2 NHLd C. albicans 30 8 7/24 7 blood cultures Dead

3 AMLe C. albicans 7.4 16 5/22 2 blood cultures, histopathology Alive

4 ALL C. parapsilosis 5.8 NA 2/12 7 blood cultures Alive

5 ALL C. parapsilosis 6.7 21 2/3 2 blood cultures, histopathology,



pre- and postmortem culture

Dead

6 ALL C. albicans 10.4 4 4/16 2 blood cultures Alive

7 ALL C. parapsilosis 30 3 3/18 2 blood cultures Alive

8 Aplastic anemia C. tropicalis 30 NA 5/13 17 blood cultures Alive

9 T-cell leukemia C. albicans 30 9 5/12 2 blood cultures, postmortem



cultures

Dead

10 ALL C. glabrata/C. albicans 5.1 3 2/2 2 blood cultures, postmortem



cultures

Dead

a Number of positive urine samples/total number of urine samples.

b ALL, acute lymphatic leukemia.

c NA, no adequately timed samples available.

d NHL, non-Hodgkin’s lymphoma.

e AML, acute myeloid leukemia.

FIG. 1. Peak D/L-arabinitol ratios obtained for the following: patients with

confirmed invasive candidiasis (group A; n 5 10), patients receiving empiric

antifungal treatment (group B; n 5 23), patients not undergoing antifungal

treatment or showing evidence of invasive candidiasis (group C; n 5 67), and

single samples from healthy nonhospitalized children (group D; n 5 56). The



broken line at the value of 4.6 corresponds to the upper cutoff limit.

FIG. 2. D/L-Arabinitol ratios for patient 2 (Table 1) with confirmed invasive

candidiasis. A, the time at which the first blood sample, culture positive for C.

albicans, was drawn. Amphotericin B was given from day 36 to day 62, when the



patient died (horizontal black bar). The broken line at the value of 4.6 corresponds

to the upper cutoff limit.

VOL. 35, 1997 D/L-ARABINITOL IN INVASIVE CANDIDIASIS 637

children showed positive D/L-arabinitol ratios 6, 10, 10, 12, 13,



14, 15, 21, 28, and 31 days, respectively, before empiric antifungal

chemotherapy was instituted, and Fig. 3 presents the

ratios over time for one of these patients. Six patients died;

four of them died during or shortly after the institution of

antifungal chemotherapy.

The remaining 67 patients received no antifungal treatment,

and the mean of their D/L-arabinitol ratios, determined by

using each patient’s peak value, was 3.2 6 1.1 (Fig. 1, group C).



Four patients had peak values above the cutoff limit, but for all

of these patients the values later returned to normal. Four

patients died due to their malignancies, and none of these had

a positive D/L-arabinitol ratio.



On the basis of the results for patients in groups A and C,

the sensitivity of the arabinitol test in diagnosing invasive candidiasis

is 100% and the specificity is 94%, resulting in a positive

predictive value of 71% and a negative predictive value of

100%. If it is assumed that all the empirically treated group B

patients did have invasive candidiasis, positive and negative

predictive values are both 85%.

Forty children were diagnosed with bacteremia. Of these

patients, 19 had at least two blood cultures positive for grampositive

bacteria, 15 patients had blood cultures positive for

gram-negative bacteria, and mixed infections were seen in 6

patients. The mean D/L-arabinitol ratio during bacteremia was

3.1 6 0.9, and one patient had a positive value of 5.2, which,



however, was recorded during a period of empiric antifungal

chemotherapy. One patient died from streptococcal bacteremia

and one died from a mixed infection (these two patients

had previously received empiric antifungal chemotherapy; see

above).

D/L-Arabinitol ratios were significantly higher during neutropenic

periods when antifungal therapy was not given (3.0 6

1.1) than during nonneutropenic periods (2.5 6 0.7) (P ,

0.01). Also, D/L-arabinitol ratios were significantly higher during

periods of bacteremia (3.1 6 0.9) than during nonneutropenic

periods (P , 0.01).



In all, a total of 1,039 blood samples for culture were drawn

during the study period. Forty-six cultures of blood from 10

patients were positive for Candida species, and 237 cultures of



blood from 40 patients were positive for bacteria. The mean

numbers of urine samples per patient delivered for D/L-ara-

TABLE 2. Peak D/L-arabinitol ratios in febrile, neutropenic children with cancer, empirically treated for invasive candidiasis



Patient no. Primary disease

Peak D/Larabinitol



ratio

Time interval (days)

between positive

D/L-arabinitol ratio



and empiric treatment

No. of positive

samples/no. of

all samplesa



Patient

status

Postmortem

culture and

histopathology

result

11 ALLb 3.2 NAc 0/21 Alive

12 AMLd 5.0 14 3/8 Alive



13 Wilms’ tumor 7.2 28 4/9 Alive

14 Non-Hodgkin’s lymphoma 7.7 NA 5/15 Alive

15 Rhabdomyosarcoma 3.7 NA 0/11 Alive

16 ALL 3.6 NA 0/36 Dead Negative

17 ALL 8.2 12 9/29 Alive

18 Megacaryocyteleukemia 4.5 NA 0/2 Alive

19 Malignant Schwannoma 4.6 10 2/18 Dead Negative

20 Rhabdomyosarcoma 2.6 NA 0/12 Dead Negative

21 B-cell lymphoma 3.4 NA 0/17 Alive

22 ALL 4.9 15 2/12 Alive

23 ALL 16.0 NA 2/5 Dead Not done

24 CMLe 3.2 NA 0/17 Alive



25 ALL 5.5 21 2/6 Alive

26 B-cell lymphoma 4.1 NA 0/8 Alive

27 B-cell lymphoma 4.3 NA 0/26 Dead Not done

28 B-cell leukemia 3.6 NA 0/9 Alive

29 AML 9.8 31 8/18 Alive

30 Neuroblastoma 4.9 13 2/5 Alive

31 AML 7.6 6 2/20 Dead Negative

32 AML 4.9 10 4/16 Alive

33 CML 2.5 NA 0/2 Alive

a Number of positive urine samples/total number of urine samples.

b ALL, acute lymphatic leukemia.

c NA, no adequately timed samples available.

d AML, acute myeloid leukemia.

e CML, chronic myeloid leukemia.

FIG. 3. D/L-Arabinitol ratios for patient 17 (Table 2), who was receiving



empiric antifungal therapy. Amphotericin B was given from day 35 to day 50

(horizontal black bar). The broken line at the value of 4.6 corresponds to the

upper cutoff limit.

638 CHRISTENSSON ET AL. J. CLIN. MICROBIOL.



binitol analysis were 15, 14, and 9 for groups A, B, and C,

respectively.

DISCUSSION


This is the first prospective study on the diagnostic value of

monitoring urine D/L-arabinitol ratios in immunocompromised



patients at high risk of invasive candidiasis. All 10 patients with

confirmed invasive candidiasis, based on multiple positive

blood cultures alone (6 patients) or such cultures together with

tissue biopsies or postmortem examination (4 patients),

showed positive D/L-arabinitol ratios (Table 1; Fig. 1). Positive



ratios were also found for 12 of 23 children who had negative

blood cultures and who were being empirically treated with

antifungal chemotherapy (Table 2; Fig. 1) and for 4 of 67

patients not receiving antifungal therapy. However, blood cultures

are usually positive for only about 50% of patients with

proven disseminated candidiasis, even when biphasic media or

lysis-centrifugation is used (1, 14, 19). It is thus tempting to

speculate that 12 of the 23 empirically treated patients with

increased D/L-arabinitol ratios had unconfirmed invasive candidiasis.



This study was not designed to monitor therapy by

using D/L-arabinitol ratios, but the data presented for two patients



(Fig. 2 and 3) indicate that the outcome of antifungal

treatment correlates with changes in D/L-arabinitol ratios.

C. glabrata has not been shown to produce D-arabinitol when



it is grown in vitro (9). The patient with both positive blood

cultures for C. glabrata and increased D/L-arabinitol ratios



probably had a mixed candidal infection, since all postmortem

tissue cultures were positive for C. albicans. Considering subjects



not included in the present study, we have also found

elevated D/L-arabinitol ratios in two nonimmunocompromised

patients with C. glabrata fungemia, but for these two patients



no tissue cultures were performed (unpublished data).

During nonneutropenic periods, children with cancer had

higher D/L-arabinitol ratios (2.5 6 0.7) than healthy nonhospitalized

children (2.0 6 0.6) (P , 0.01). The ratios further

increased (3.0 6 1.1) during neutropenic periods without empiric



antifungal chemotherapy. This could reflect an increased

fungal load in these patients at risk for fungal infection, although

fungal surveillance cultures were not included in our

study, and all patients were given nystatin orally. D/L-Arabinitol

ratios also increased (3.1 6 0.9) during bacteremia, and it has



previously been reported that bacteremia is a risk factor for

fungal infection in patients with hematologic disorders (7). We

do not believe, however, that bacteremia per se gives rise to

increased D/L-arabinitol ratios in urine, since we have found

normal D/L-arabinitol ratios in all of 25 adult nonimmunocompromised



patients with bacteremia whom we studied (unpublished

data).

Perhaps the most striking finding of this study is that D/Larabinitol



ratios often increased days to weeks before invasive

candidiasis was suspected or confirmed through blood cultures

or histopathology. Elevated D/L-arabinitol ratios were found 3



to 31 days (median, 12 days) before a culture-positive blood

sample was first drawn from 7 of 10 children with confirmed

invasive candidiasis and before empiric antifungal treatment

was initiated for 10 of 12 children. The time gain achieved with

D/L-arabinitol ratios was actually even greater considering the



time needed for culture. It has previously been reported that

the outcome for patients with invasive candidiasis depends

largely on the early institution of antifungal therapy (2, 15).

Renal dysfunction influences the absolute levels of D-arabinitol



in both serum and urine (18). The method of using

D/L-arabinitol ratios, which was first suggested by Roboz et al.



(18), has been found to overcome this problem, and, using

negative chemical ionization mass spectrometry, Roboz and

Katz (17) found increased D/L-arabinitol ratios in serum from



15 of 16 patients with confirmed candidiasis. The present study

indicates that monitoring of D/L-arabinitol ratios in the urine of



immunocompromised children with cancer is useful for diagnosing

invasive candidiasis. A considerable advantage of our

method is that the analysis can be performed on a standard gas

chromatograph, which expands the diagnostic applicability of

D/L-arabinitol ratio measurements. It should be emphasized



that the method has not yet been evaluated prospectively with

immunocompromised adults or multitrauma patients with

Candida superinfections due to long-term broad-spectrum antibiotic

treatment. Nevertheless, we have found increased D/Larabinitol



ratios in urine samples from several such patients

(unpublished data).

Another approach to solving the problem of increased concentrations

of D-arabinitol in the serum of patients with renal

dysfunction is to determine the D-arabinitol/creatinine ratio. In

a prospective study, Walsh et al. (22) found elevated D-arabinitol/



creatinine ratios in 74% of cancer patients with verified

candidemia and in 40% of patients with deep-tissue candidiasis

without fungemia. Although they used another method to detect

the in vivo production of D-arabinitol, the results of Walsh



et al. (22) support our unpublished observations that increased

D/L-arabinitol ratios in urine are seen also in adult cancer



patients with invasive candidiasis.

A number of circulating candidal antigens, e.g., the cytoplasmic

Candida enolase antigen, cell wall mannan, and heat-labile



glycoprotein antigens (Cand-Tec), have also been used for

diagnosis (5). Analysis of multiple serial serum samples may

improve the sensitivity of Candida detection, since antigens are



cleared rapidly from serum and/or immune complexes are

formed. Walsh et al. (20) found a diagnostic sensitivity of 75%

and a specificity of 96% in a prospective clinical trial by using

multiple sampling and an assay for Candida enolase in serum.



Studies on the detection of serum mannan have shown sensitivities

from 0 to 65%, as reviewed by de Repentigny (5).

Antibody tests are often negative for immunocompromised

patients, and it has been claimed that such tests are of more

prognostic than diagnostic significance (11). Nevertheless, Deventer

et al. (6) found anti-Candida enolase antibodies in 53%



of a group of immunodeficient patients with invasive candidiasis,

and Navarro et al. (12) reported a sensitivity of 89% when

analyzing antibodies to cell wall-bound and cytoplasmic antigens.

It should be pointed out, however, that neither of the

cited reports presented any data regarding antibody kinetics

during the course of infection. Newer techniques such as Candida



DNA amplification have produced encouraging results in

small studies, and sensitivities of between 60 and 79% for

culture-positive clinical specimens have been achieved (3, 4, 8).

Another approach involves a Limulus amoebocyte lysate assay

for the detection of elevated levels of 1,3-b-D-glucan in plasma,



which was found to have a sensitivity of 90% for patients with

confirmed fungal infection (13). To date, however, neither of

these two methods has been thoroughly evaluated in prospective

clinical studies.

The lack of a “gold standard” for the diagnosis of invasive

candidiasis complicates the evaluation of new diagnostic methods.

However, the results of the present study indicate that

elevated D/L-arabinitol ratios can be detected and used as a



valuable predictor at an early stage of invasive candidiasis in

children with cancer. Therefore, we suggest that, together with

routine blood and tissue cultures, the D/L-arabinitol ratios for



such patients should be monitored routinely, and for patients

with increased ratios, antifungal chemotherapy should be instituted.

VOL. 35, 1997 D/L-ARABINITOL IN INVASIVE CANDIDIASIS 639




ACKNOWLEDGMENTS


This study was supported by grants from the Children’s Cancer

Foundation of Sweden, the Royal Physiographic Society in Lund, and

the Medical Faculty of Lund University.

REFERENCES

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