Wednesday, 23 July 2014

If Candida is at epidemic levels is Thrush also

August 10, 2012

Headaches. Recurring heartburn. Lethargy and fatigue. Prostatitis. Night sweats. Diarrhea. Fibromyalgia. Sinusitis. PMS. Anxiety. “Brain fog." Memory loss and mood swings. These are just a few of the symptoms that manifest a condition known as candidemia (Candida in the blood). Some researchers believe that Candida albicans is the leading causative pathogen behind a host of medical conditions plaguing Americans today. Understanding the role of Candida albicans in a normal functioning digestive tract and the havoc it can wage on the body’s immune system can help you stir clear of this hidden invader.

More Photos

Rarely diagnosed
As you can see from the list of symptoms above why a physician might easily overlook this condition, especially if only few of the symptoms are present. This helps to explain why some have estimated that as much as 80 percent of Americans have some form of Candida overgrowth. This is largely due to the widespread use of antibiotics in treating infection, and as a prophylaxis. With the exception oral and vaginal candidiasis (a Candida infection), which can be visually diagnosed, most forms Candida overgrowth go undiagnosed. This helps to explain why a severe form of Candida overgrowth known as invasive candidiasis can be life threatening if it left undiagnosed and untreated.
What is Candida albicans?
Candida albicans is a ubiquitous fungus commonly found in the mucus membranes of the digestive tract, mouth, and genital region. Normally Candida plays a commensal role in the flora of the digestive tract where it is balanced by beneficial bacteria. However, if this delicate balance is upset in anyway, e.g., the use of antibiotics or a compromised immune system, Candida can thrive and overwhelm the system. If the Candida is not brought back into balance in the GI tract it can penetrate the mucosal wall and enter the bloodstream where it can wreak havoc on any number of vital organs and anatomical structures, including the heart, brain kidneys, prostate and liver. Candida albicans is also one of the principle agents responsible for decomposition of the body after death.
A delicate balance
In its normal yeast state Candida albicans takes on a harmless oval shape. While in this form it serves a very beneficial role in the digestive tract aiding in the decomposition of food. To help keep Candida in balance the intestinal flora also contains acidophilus and bifidophilus bacteria that secrete substances that inhibit the overgrowth of Candida. Before the 1940s Candida infections were a rare occurrence. Suddenly, however, with the advent and widespread use of antibiotics in the early 1950s there appeared a tidal wave of women's yeast infections. “Well, first, obviously, the introduction of antibiotics paralleled the rise in yeast infections. In fact initially a prescription for antibiotics directly preceded almost all yeast infections. Yeast infections followed a course of antibiotics by a few weeks,” cautions Dr. Jerry Glenn Knox BA, DC.
Today the epidemic continues as more and more cases of invasive candidiasis and candidemia plague hospitals. According to the CDC (Center for Disease Control and Prevention), “Hospital-associated infections such as candidemia are a leading cause of bloodstream infections in the United States. Advancements and changes in healthcare practices can provide opportunities for new and drug-resistant fungi to emerge in hospital settings.”
Antibotics and Candida
Broad spectrum antibiotics such as Amoxicillin, Ampicillin or Levofloxacin can have diarrhea as a possible side effect. The reason for this is simple; in addition to killing off the unwanted microorganism attacking your body, antibiotics also target the beneficial bacteria in your gut which can result in a bout of diarrhea. The diarrhea is an indication that your intestinal flora is out of balance and unable to process food properly. Usually the diarrhea generally abates after completing the course of antibiotics. However, this can become problematic in cases where antibiotics are prescribed for an extended period of time—three weeks or more.
This was the case with a 57-year-old gentleman in 2004. After undergoing an ultrasound-guided transrectal biopsy of his prostate to investigate an elevated PSA, he was placed on fluoroquinolone (an antibiotic) to minimize risk of infection following the procedure. His biopsy tested negative for malignant cells. One week following the procedure he developed urinary retention and was catheterized and placed on Tamsulosin with positive results. About three months later while on a business trip he experienced painful urination and was prescribed antibiotics. Unfortunately, the symptoms continued so he reported back to his urologist. A urinalysis was performed but cultures were negative. He was instructed to continue his antibiotics. After one week he again experience urinary retention and underwent catheterization. Additonally, his Tamsulosin was increased and antibiotic regimen continued. A second urine culture, without urinalysis, was taken three days later and returned negative findings. Though the symptoms never completely resolved, the patient never experienced systemic indicators like fevers or chills.
After one month of antibiotics, his dysuria progressed to perineal discomfort and burning upon urination. Further examination by the urologist revealed a slightly boggy prostate along with mild tenderness. Results from urine, prostate secretion and ejaculate cultures at this time all grew Candida albicans. Based on these new findings, the patient was diagnosed with Candida prostatitis and was started on an antifungal medication (Fluconazole) daily for 6 weeks. The patient saw resolution of symptoms after the first week of therapy. He remained symptom-free over a year later, and follow-up urinalysis following treatment has returned to normal.
Hard to find
Doctors routinely prescribe antibiotics to treat infectious disease. There are a variety of antibiotics on the market designed to treat specific types of infectious microorganisms and they are best prescribed when the specific infection-causing pathogen can be identified. Unfortunately the lab work necessary to properly indentify a specific causative pathogen before prescribing an appropriate antibiotic treatment is time consuming; yeast cultures may take up to 30 days to yield results. Since immediate palliative patient care is a doctor’s primary role, broad spectrum antibiotics are routinely administered to treat infectious disease. Unfortunately, these treatments also target the “friendly bacteria” in the gut which creates a favorable environment in which Candida can thrive. Left unchecked, Candida can penetrate the intestinal mucosa and enter the bloodstream and wreck havoc on the body, especially immunocompromised individuals with HIV.
Battling Candida overgrowth
The key to waging any successful military campaign is knowing your enemy’s strengths, weaknesses, allies and enemies. This same principle applies to battling Candida overgrowth. In our next article we will examine each of these four factors to help equip you to wage a successful four-prong campaign against Candida overgrowth.
The mission
There is a literal tsunami of information about Candida albicans on the internet and it is easy to find yourself buried under it. The goal here was to provide a fundamental understanding about Candida albicans which will be supplemented by future articles. My mission is to distill and decipher the latest news and information about the treatment of Candida-related ailments into a readily digestible format that is easy to process and utilize for your own well being.
This information is not intended as professional medical advice or is it an alternative to a doctor’s care. Caution should be exercised in using any self-help remedies to treat Candida overgrowth without a thorough understanding of the benefits and potential side effects. In either case, seek prompt medical care if you note any usual changes in your health and bodily functions.
________________________________________________________________
Mr. Collins, a cancer survivor, has first-hand experience fighting Candida overgrowth. A former publisher of the New York edition of Doctor of Dentistry magazine, Henry has completed certificate courses in anatomy, medical terminology, physiology and pathophysiology. He is currently writing a book on prostate cancer and is an advocate of early PSA screening. He can be reached at: info@patientmanual.com.

Tuesday, 22 July 2014

Kill your Candida and lose weight

One of the symptoms of systemic Candida is weight gain, or difficulty losing weight. It can cause the kind of stubborn fat deposits that are hard to shake off, no matter how little you eat or how much exercise you do.
There are a couple of reasons for this, but first it’s important to understand that no diet is going to help. Treating the underlying cause of the problem – the Candida overgrowth – is the way to get back in shape. Candida can lead to excess fat deposits in a few different ways. Read on below to find out more.

Candida Toxins

Candida cells are constantly reproducing and dying. This does not happen only during Candida Die-Off. Even if you are not treating your Candida at all, the natural life cycle of this yeast means that toxins from dying Candida cells are constantly being released into your bloodstream. Why does this matter? Well, your liver has to process these toxins and expel them from your body. If your liver becomes overloaded (i.e. there are too many toxins in your bloodstream), then it has to store these nasty chemicals somewhere else for processing later. Your liver does this by storing them in fat cells, primarily around the hips, belly and thighs. For many dieters, this is the root cause of those abnormal fat deposits.

Sugar Cravings

Candida needs sugar to grow and reproduce, sugar that comes from the foods we eat. A typical symptom of a Candida infestation is that the patient is eating lots of sugar and carbs but still craving more. In Candida sufferers, this is far more than just a psychological addiction to sugar. The Candida yeast is processing large amounts of sugar and sending your blood sugar levels lower, triggering signals from your brain that you need to eat more. This is one way that Candida can cause overeating.

Stress On Your Immune System

The toxic byproducts of Candida can trigger a response from your immune system. Stress on the adrenal glands raises cortisol levels, an emergency response which prompts your body to hold on to every last piece of fat that it can.

Fatigue

Here’s one cause of weight gain that almost every Candida sufferer will identify with. The Candida cells release up to 79 different toxins, including a particularly nasty neurotoxin named Acetaldehyde. These toxins can cause symptoms like brain fog, fatigue and depression. Because of this, Candida sufferers often find it hard to get the exercise that they need to stay healthy and in shape.
Often a simple calorie restriction diet is treating the symptom rather than the cause of the problem. Diet advice is everywhere on the internet, but rarely do you see a treatment plan that can treat the underlying condition that is causing the weight gain. By following a good Candida treatment plan and sticking to a healthy diet, you might find it easier than you think to shake off those stubborn fat deposits

CCSVI-MS- too much money making - when will they wake up!!!!!!!!!!!!!!!!!and the MS Society justs sits by

CCSVI is big money making business, MS is a big money making business especially to drug producing companies, CCSVI in MS patients has been known for years they have been  having stents fitted at their own expense for years. But the cause of this has been ignored even by the MS Society. I know CCSVI is caused by a misaligned Atlas and MS does NOT even exist. How many millions and for how many years are they going to waste and how many lives will they ruin until they admit how valuable a trained Chiropractor's opinion is
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CCSVI - research so far and next steps


So far, many studies have been published looking at how prevalent CCSVI is in people with MS, and also looking at the effects of treatment for CCSVI.  Although none of the studies have been conclusive to date, more research is constantly being published that will shine a light on the potential relationship between CCSVI and MS.misaligned Atlas

Studies on prevalence

Many studies looking at the link between CCSVI and MS have been published.  Some of these studies show a strong link between CCSVI and MS, and others show no link. This is likely to be caused by:
  • the differences in techniques used to detect CCSVI
  • different study sizes
  • the way the study was conducted.
Some of these studies have highlighted the need for standardised techniques when it comes to diagnosing and measuring CCSVI, as well as robust study design, in order to measure the true prevalence of CCSVI in people with MS.  Research is underway to address these issues for future studies.

Buffalo study

One of the largest studies to date on the prevalence of CCSVI was carried out in Buffalo NY.
500 people were scanned by doppler scanning methods to detect signs of CCSVI:
  • 56.1 per cent of 289 people with MS showed signs of CCSVI
  • 22.7 per cent of 163 people without MS (healthy participants) also showed signs of CCSVI
  • 38.1 per cent of 21 people with clinically isolated syndrome showed signs of CCSVI
  • 42.3 per cent of 26 people who had other neurological conditions showed signs of CCSVI
Looking at this initial data, the study organisers have said these results suggest that CCSVI does not have a primary role in causing MS, but may be a result of MS.
1000 additional participants will now be examined for signs of CCSVI by more advanced screening methods in the second stage of the study, to get a more accurate picture of the prevalence of CCSVI in people with and without MS.
Based on these results, a small clinical trial involving 30 people with CCSVI is underway. 

Italian study

Another large study looking at 1165 people with MS, 376 healthy volunteers and 226 people with other neurological conditions reported back in October 2012.
CCSVI was found in:
  • 3.26% of people with MS
  • 2.13% of healthy volunteers
  • 3.1% of people with other neurological conditions
The researchers conducting the study, which is yet to be published in a peer-reviewed journal, said there were no statistically significant differences in the three groups.

Seven key NMSS funded CCSVI studies

In June 2010, the National MS Society and the MS Society of Canada announced $2.4 million of support for seven new research projects focusing on the role of CCSVI in MS.
Results from these seven key studies have started to be published.

Texas study

Researchers from the University of Texas Health Science Centre at Houston used various neurosonography techniques to measure blood outflow from the brain to establish whether any of the participants in the study fulfilled the criteria for CCSVI. As part of their study they replicated the ultrasound methods used in the original studies which identified CCSVI in people with MS.
Researchers assessed CCSVI in:
  • 206 people with MS
  • 70 people without MS
The researchers found that CCSVI was present in 3.88% of people with MS and 7.14% of people without, and that there were no significant differences in blood flow rates between people with MS and people without.

Canadian study

Researchers from the University of British Columbia and the University of Saskatchewan in Canada used ultrasound techniques and catheter venography - as used in the original CCSVI studies - to ascertain whether the study participants had CCSVI.
177 people took part in the study (79 with MS, 55 of their siblings and 43 unrelated people who didn’t have MS) across three different centres in Canada.
Using catheter venography the researchers found that the following fulfilled criteria for CCSVI:
  • 2% of people with MS
  • 2% of their siblings
  • 3% of unrelated people without MS
Researchers also used ultrasound to look for evidence of CCSVI and found the following had signs of CCSVI:
  • 44% of people with MS
  • 31% of their siblings
  • 45% of unrelated people without MS
Both sets of results showed no significant increase in prevalence of CCSVI in people with MS compared to people without MS.

Interventional studies

In addition to studies on prevalence, there have also been a few small scale clinical trials looking at the safety and effectiveness of treatment of CCSVI on people with MS.
These studies focused mainly on safety of the treatment. They also looked at things like relapse rate, disability status and also measured lesions in the brain on an MRI machine, but these studies were open labeled, meaning that placebo effects were not accounted for. It remains difficult to draw firm conclusions on the potential risks and benefits of CCSVI treatment in people with MS without doing larger, more controlled studies. 
There are a number of studies that have looked at the risk of treatment for CCSVI. Some complications that have been described are:
  • developing clots in the vein
  • arrhythmias (disruptions to the heart beat and rhythm)
  • vein dissection (separation of the vein)
  • in-stent thrombosis (bleeding at the site of stent placement)
  • vein rupture
  • groin hematoma (collection of blood outside the blood vessel).
In May 2012 the US Food and Drug Administration (FDA) issued a safety communication on CCSVI which mentions reports of adverse events associated with treatment for CCSVI.
Larger randomised controlled trials looking at the safety and effectiveness of treatments for CCSVI in people with MS will hopefully give us a more accurate picture of the potential risks and benefits of treatment for CCSVI. 
Until then, we do not recommend that people seek treatment for CCSVI outside of a properly regulated clinical trial.
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Sunday, 20 July 2014

CANDIDA causes the body to decompose as we are alive??????????

Candida causes diabetes, asthma, eczema, allergies, IBS , joint pain, acid reflux, nail fungus, toxicity of the liver, problems with the heart and lungs, thrush.

We are so busy buying over the counter remedies accepting these ailments as the norm, that we are missing the point, which is the reason- CANDIDA

I believe that the  body is trying to decompose, as when we die so does the good bacteria, that's why diabetes is so common as so is cancer, we are breaking down as we are alive.

Friday, 18 July 2014

everyone should watch this video

http://www.atlantotec.com/en

misalinment of the Atlas affects the BRAIN


Stay healthy without continuous medication?

Here you will learn how migraine, whiplash, neck pain, back pain and dozens of other complaints can be cured by adjusting the position of the Atlas.


Schädel mit Atlaswirbel für Atlaskorrektur gegen Migräne und Schwindel
Atlas vertebra:
Die Atlaswirbel-Fehlrotation kann Schwindel und Migräne auslösen

  • Do you want to be rid of your complaints for good? If so, stop fighting only the symptoms!

  • One single treatment and a follow-up check will activate the self-healing power of
    your body.

  • 9 facts you should know before undergoing other
    kinds of treatment.

Read on to find out why misalignment of the first cervical vertebra may be responsible for a whole series of apparently unrelated problems, the treatment of which focuses merely on suppressing the pain without removing the cause. A new discovery could now bring relief in many cases.

Atlas correction

The Atlas is the hidden cause of numerous complaints

The first cervical vertebra, known as the Atlas or C1, plays the most delicate and at the same time primary role for the entire spine.

A malfunction in this area can produce a "domino effect" on the entire musculoskeletal system, as well as the circulation and nervous systems, creating imbalances and dysfunctions in various parts of the body.

With time these dysfunctions may turn into serious health problems of apparently unknown origin. The body is not able to function as it should, seriously affecting quality of life, yet no organic cause can be identified or diagnosed.


Migräne Kopfschmerzen psychosomatisch

Are your complaints really just psychosomatic?

How many patients are told by their doctors that their problems are unknown and that they "obviously" have a psychosomatic origin? It is quite likely that your doctor doesn't know – and therefore doesn't tell you – that a misaligned Atlas might be the cause of your problems.

Chronic complaints after whiplash trauma and recurring migraine attacks or headaches find a simple, logical explanation in the Atlas misalignment. Correction of this problem offers a real, lasting solution without being forced to take medicine continuously with the ensuing side effects.

In addition, correction of the Atlas vertebra is the ideal method to improve posture, thus preventing postural defects such as functional short leg, pelvic obliquity, hyperlordosis/hollow back, kyphosis, idiopathic / functional scoliosis and hypolordosis.

9 Facts you should know about misalignment of the Atlas vertebra

Migräne
  • For many people the trauma of birth leads to a misalignment of the Atlas (first cervical vertebra) and sometimes also of the Axis (second vertebra).
  • Traumas such as whiplash or falls may also cause or further accentuate misalignment of the Atlas vertebra.
  • It has been found that misalignment of the Atlas is responsible for a number of disorders and conditions at both the physical and mental level.
  • Conventional medicine does not consider misalignment of the Atlas vertebra a possible cause of disorders in the patient. As a result this problem generally goes undiagnosed.
  • Failure to diagnose the problem is due to the fact that misalignment of the Atlas does not show up in regular X-rays or magnetic-resonance imaging.
  • A multi-slice helical CT scan allows misalignment to be detected clearly and measured, but only if the radiologist sets up the CT machine for this purpose.
  • Leaving aside severe dislocations of the vertebrae requiring surgery, conventional medicine offers no treatment to correct the misalignment of the first cervical vertebra.
  • A single ATLANTOtec® session is all that is needed to permanently return the Atlas to the correct position.
  • Many consider that acting on the neck area is dangerous. You should know that any potential danger is limited to chiropractic manipulations. The ATLANTOtec® method uses a completely new, riskfree principle, which has nothing to do with chiropractic, osteopathy or any other familiar methods.

The most common complaints which have benefited from realignment of the Atlas vertebra

The negative effects of misalignment of the Atlas may manifest themselves throughout the entire body.

Data collected and records of patients treated with ATLANTOtec® have enabled us to draw up a list of the most common complaints and symptoms which may be alleviated by repositioning the Atlas. It is important to understand that not all disorders listed disappear in anyone who undergoes Atlas treatment, as they may have other causes or contributing factors.

It would be wrong to interpret correction of the Atlas as a miracle cure that can solve all problems. On the contrary, it is essential to understand that the purpose of ATLANTOtec® absolutely does not consist in diagnosing or "curing" a variety of disorders, but simply in seeing whether it is necessary to treat the Atlas. What is miraculous is the body's ability to self-heal, once put in a position to do so!

Correcting the position of the Atlas, if misaligned, is necessary, irrespective of any problems temporarily experienced, as such treatment is an "investment" in good future health. Realignment is advisable starting with children and allows the body to function at its full potential.
Beschwerden
  1. migraine
  2. headache
  3. chronic pain following whiplash
  4. vertigo – dizziness – unsteadiness
  5. Ménière´s disease
  6. trigeminal neuralgia
  7. temporomandibular joint syndrome
  8. chronic sinusitis
  9. asthma (may have other causes)
  10. limited or painful head rotation or bending
  11. cervical pain
  12. stiff neck – torticollis
  13. shoulder pain / one shoulder higher
  14. formication / numbness of the arms
  15. recurrent tendinitis
  16. tennis elbow / carpal tunnel syndrome
  17. permanent muscle tension
  18. chronic muscle pain
  19. back pain
  20. idiopathic scoliosis
  21. lumbago
  22. herniated disc – disc disease – protrusion of discs
  23. compressed spinal nerves
  24. asymmetry of the pelvis
  25. arthritis – osteoarthritis(if arising from poor posture)
  26. blocking of the sacroiliac joint
  27. inflammation of the ischiadic nerve – sciatica
  28. hip pain
  29. functional leg length difference
  30. pain in the legs, knees or feet
  31. tendency to have cold hands / feet
  32. chronic eye inflammation / vision disorders
  33. tinnitus (buzzing or ringing in the ears)
  34. chronic middle ear inflammation
  35. abnormal heart rhythm / altered blood pressure
  36. digestive problems / gastric hyperacidity
  37. gastric reflux
  38. chronic diarrhea or constipation
  39. depression (if associated with chronic pain)
  40. insomnia / falling asleep
  41. chronic fatigue
  42. fatigue syndrome (CFS) (also by acidosis)
  43. allergies / hay fever (improvements for unknown reason)
  44. learning difficulties / dyslexia
  45. epilepsy

Migraine from a medical point of view

Migräne Kopfschmerzen Kopfweh
Generally, patients suffering from headaches or migraines typically go to the doctor thinking that they will be CURED. They soon discover that their doctor's concept of cure is different from their own.

During their university studies, doctors learn that treating migraine, for example, involves relieving symptoms or pain, and not seeking the cause of the symptom itself in order to cure it, thus preventing the same problem from recurring in the future.

Doctors examine patients and collect data with the sole purpose of prescribing the "best" medicines manufactured by pharmaceutical companies and promoted in their surgeries in order to achieve sales.

The modern doctor has turned into a veritable "drug pusher"! The reason is a lack of interest on the part of the pharmaceutical industry in finding a permanent solution for migraine, as a lifetime patient is a lot more profitable than a healed one. Moreover, doctors must adhere to guidelines while treating their patients and therefore are committed to prescribing pills rather than alternative treatments.

If you don't believe it, ask yourself the following question: when was the last time the pharmaceutical industry announced that it had found the definitive cure for any given disease?
Those who become aware of this fact stop running to their doctor to try to solve their chronic illnesses such as migraine or headache, but instead keep on searching for alternative solutions.

Before patients come to this conclusion, unfortunately they need to fall flat on their face. Only after suffering a pill-induced gastric ulcer and endless migraine and headache attacks do they start searching for alternative solutions.

In an emergency a pill can be useful. However, it does not represent a permanent solution for constantly recurring migraines or headaches!

Ursache Migräne

Atlas misalignment as a possible reason for migraine

Misalignment of the Atlas does not only compress the nerves but also the adjacent arteries and veins. This results in a circulation disturbance of the brain, which in combination with other factors may lead to migraine and headaches as well as various other health problems.

The theory that migraine is a neurological disease melts away like the snow in the sun after a correction of the Atlas! (See also this study)


Each Atlas adjustment is not like any other Atlas adjustment

There are several methods of correction of the Atlas: AtlasPROfilax, Atlaslogy, chiropractic, Atlas-Orthogonal and Upper Cervical. The theories and results are highly dissimilar. Read more in the following article (German): Each Atlas adjustment is not like any other Atlas adjustment.

Correction of Atlas and cessation of a seizure disorder

CESSATION OF A SEIZURE DISORDER: Correction of the Atlas Subluxation Complex

Robert J. Goodman, D.C., John S. Mosby Jr., D.C., M.D.

ABSTRACT

Observations of one patient presenting with a seizure disorder are reported. Relief of symptoms is

noted subsequent to correction of the misalignment of the occipito-atlanto-axial complex. The authors

suggest a relationship between the misaligned skull and subjacent vertebrae and some seizure disorders.

Key Words: epilepsy; atlanto-occipital joint

INTRODUCTION

The term epilepsy refers to any disorder characterized by recurrent seizures. Seizures are transient

disturbances of cerebral function due to abnormal paroxysmal neuronal discharges in the brain.

Approximately 0.5% of the U.S. population is affected. 1, 2



Epilepsy is grouped into two different etiological categories, idiopathic or constitutional and

symptomatic epilepsy. In idiopathic or constitutional epilepsy, seizures usually begin between 5 and 20

years of age. NO specific cause can be identified, and there are no other neurological abnormalities.1 The



causes of symptomatic epilepsy include abnormalities and perinatal injuries, disorders of metabolism,

trauma, space-occupying lesions, vascular problems, degenerative disorders, and infectious diseases. 1, 3-6



Clinically, seizures are categorized by description. The two major descriptive classifications are

partial seizures and generalized seizures. Partial seizures are determined by clinical observation and by

electroencephalograph manifestations. They affect only a restricted part of one cerebral hemisphere. In

simple partial seizures the patient remains conscious, but in complex partial seizures consciousness is lost.

Partial seizures may evolve into generalized seizures.1



Generalized seizures are categorized as petit mal or absence seizures, atypical seizures, myclonic

seizures, akinetic seizures, grand mal or tonic-clonic seizures, atonic or ionic seizures, and seizures that will

not fit into any other category.

Absence or petit mal seizures cause some reduction in postural tone, with some clonic or tonic

components. Consciousness is impaired. These attacks occur quickly. When the attack occurs as a person

is speaking, the person may miss a few words in mid-sentence then resume with the remainder of the

sentence when the attack subsides. If a child has a petit mal seizure while playing, for instance, he or she

may freeze, that is, stand perfectly still while reaching for a toy. These seizures will often cease at

approximately 20 years of age. Diagnosis is assisted by electroencephalographic studies which show

bilateral synchronous and symmetric 3Hz spike-and-wave activity.1



Atypical seizures are almost identical to the petit mal seizure except that changes in tone are more

dramatic and onset and termination of the attack is slower.1



Myclonic seizures cause single or multiple myclonic jerks and myofacial spikes are seen on an

EEG.1 Akinetic seizures present as a sudden loss of consciousness and EEG findings show synchronous

firing from deep lesions often in the frontal regions of the brain.7



The grand-mal or tonic-clonic seizure occurs with a sudden loss of consciousness, the patient

becomes rigid then falls to the ground and respiration is arrested for less than 60 seconds. This is described

as the tonic phase. The next phase is the clonic phase during which the body jerks violently for 2 to 3

minutes. Flaccid coma occurs next. During this type of seizure the tongue may be bitten and urinary or

fecal continence may be lost. The patient will then either recover consciousness, drift into sleep, or never

recover consciousness which is called status epilepticus.1



Atonic seizures are epileptic drop attacks where the patient, usually a child, loses all motor tone

and falls to the ground.8 The stigma of the helmet is often conferred of necessity on the patient with drop



attacks in order to protect him from further injury during these ictal events. These spells are often

intractable to treatment. Finally, the onset of ictal falling almost always occurs in patients who already

have one of more other types of seizures and implies a poor prognosis for the ultimate seizure control and

for normal mental development.9-11



Lennox-Gestaut Syndrome (L-G) is another classification of seizure disorder which has a peak age

of onset of four years.12 It is characterized by myoclonic and atypical absence seizures, regression of



intellectual functions and generalized spike wave discharges on the EEG at a rate below what is seen in

petit mal seizures. Several hundred attacks may occur in a day and multiple injuries from falls are

common. Diffuse cerebral atrophy is seen in 60% of cases.8



MEDICAL TREATMENT

The descriptive classifications above are important for determining the most appropriate medical

treatment. When patients have recurrent seizures, medication is prescribed until there have been no

seizures for at least four years. Epileptic patients are advised to avoid situations that could be dangerous or

life-threatening during seizures.1



All doctors must be aware of a condition called status epilepticus which is the rapid succession of

seizures so that the next seizure begins before the previous one has ended. Status epilepticus is a medical

emergency since continuous epileptic activity can damage the brain permanently. Of course death may

occur during a seizure if the patient aspirates contents of the stomach which either occlude airways or

prevent adequate oxygenation.8



When patients have been seizure free for at least four years withdrawal of medication may be

considered. There is unfortunately no way of predicting which patients can be managed without treatment.

Recurrence of attacks is most likely in patients who initially fail to respond to therapy, those with

convulsive jerking movements, those with multiple types, and those with continuing EEG abnormalities.1



HISTORY

Patient E is a five year old white female, who was small at birth and born breech. During her first

three years of life she had many viral infections and repeated attacks of otitis media. Some concern was

expressed by her mother over growth and language retardation. At age 13 months, however, psychomotor

skills had been assessed as normal.

At the age of 4 years 8 months, in October 1988, Patient E was playing as a day care center, when

she struck her head on the underside of a table. Within two hours her first grand mal seizure occurred.

Patient E’s second seizure occurred within three weeks.

Patient E was evaluated at the Mayo Clinic and was experiencing 10 to 30 seizures per day with

no seizure-free days. (Figure 1 is a graph prepared from Patients E’s daily seizure journal kept by her

parents. The recorded the number, time, type and severity of each seizure.) Seizure types were described as

being tonic, clonic, akinetic, and grand mal. The diagnosis of Lennox-Gestaut Syndrome was made. The

patient was described as being able to speak only a few intelligible phrases. She was tremulous, and had

difficulty standing.

Various laboratory tests were performed including a complete blood count, serum ammonia and an

SMA 20 and all were within normal ranges. A CT scan and MRI were negative for fractures and

pathologies. The EEG, however, showed a slow spike-wave abnormality as well as other features which

may occur in Lennox-Gestaut syndrome. The prognosis was determined to be grave and the comment was

made that the L-G syndrome is fraught with nearly uniform disappointment since medical therapy has very

few beneficial results.

Patient E was given Depakote, Zarontine, and ACTH therapy. Each was given singly and then

discontinued due to Patient E’s unusual or aggressive behavior. Finally, Tegretol/carbamazepine seemed to

have some positive effects on the grand mal type seizures. It was after this, however, that Patient E began

to have drop attacks.

On July 18, 1989 Patient E entered into a team evaluation and treatment plan at the Palmer

Chiropractic Clinic. At that time, Patient E was having 30 to 70 seizures per day. {Figure 1] Patient E

presented with a helmet, faceplate, and harness for protection against falls.

The patient’s communication skills were retarded. Physician exam revealed a waxy buildup in the

ears so that the TM could not be visualized. Neurological exam revealed the presence of hyper reflexive

and asymmetric reflexes. Orthopedic evaluation was unremarkable.

Further chiropractic evaluation revealed paravertebral muscular spasm in the cervical area;

cervical ROM severely restricted especially in the right lateral bending; right leg deficiency of �� to 1 inch;



suspected cervical misalignment resulting in an atlas subluxation complex.

A specific upper cervical x-ray series was taken.13,14 During patient placement for the nasium



view, certain postural deviations were noted. When asked to sit up straight, Patient E’s head, cervical, and

upper thoracic spine would not center directly over the pelvis and demonstrated excursion into the frontal

plane. No pathologies were noted on the x-rays.

Chiropractic x-ray analysis revealed a misalignment of the occipito-atlanto-axial region. Figure 2

is a representation of a radiograph showing Patient E’s misalignment in the frontal plane view. Rotation of

atlas in the transverse plane was also measured, but was minimal.

To correct the subluxation, Patient E was placed on the adjustment table side posture with the C1

transverse process as the contact point. An adjusting force was introduced to the spine using specific upper

cervical adjusting procedures.15 The success of the adjustment was measured by lessening of leg disparity,



increase of pelvic resistance, postural changes, and finally post adjustment x-ray analysis.

Patient E was adjusted on three consecutive days. After the first adjustment, Patient E’s right leg

changed from one inch deficiency to no noted deficiency.16 The patient began to rub her eyes and seemed



drowsy after the adjustment. The seizure pattern remained quite high that day. On the second day, the preadjustment

leg deficiency was 1/4th inch on the right. After the adjustment, the legs were even and the



pelvic resistance on the right was stronger that the day before. The adjustments were always made between

9:00 am and 1:00 pm. After 1:00 pm on the second day, Patient E had no more seizures during the day.

On the third and final day of care at the Palmer Chiropractic Clinic, the leg deficiency was 1/8th inch.



Again, after the adjustment, the legs were even, and the pelvic resistance was equal bilaterally. Patient E

had no further seizures after 1:00 pm on this day. The parents of Patient E commented that she was

showing more energy and more stability when standing. A postural change was quite evident when Patient

E was prepared for the post x-ray series. When instructed to sit up straight, Patient E’s head and upper

spine were centered over her pelvis. The vertical centering lines on the bucky were used for comparison.

Post x-ray analysis revealed a 91% reduction of the misalignment factors with all structures centered on the

vertical axis.

The parents had been counseled that after an adjustment they could expect exacerbations or

changes in symptomatology on the 3rd, 7th, 14th, and 28th day.17 On the 17th day after the adjustment

procedure, the seizures numbered almost 100 (more than ever before). On the 27th day, the seizures abated.



[Figure3] The seizures remained absent for approximately four weeks. The carbamazepine dosage was not

changed during this period.

At the time of this report, the dosage had been reduced by almost one half for the past two weeks.

Patient E has had six or fewer seizures per day. The seizures are completely absent on some days. After

fewer than 60 days of chiropractic care Patient E has been speaking with five or six word sentences. A

current speech evaluation is pending from her therapist and our patient has not been adjusted since July

1989. At re-evaluation, Patient E had no leg deficiency and her condition continues to improve.

DISCUSSION

The authors could find little chiropractic literature pertaining to epilepsy or seizure disorders.

Young reported on three cases which responded to chiropractic care.18 IN the medical articles reviewed on



this subject, little hope, if any, is expressed for the control or cessation of childhood seizures of this type.

Using statements like “woefully”, “poor diagnosis”, “retractability to treatment”, “fraught with uniform

disappointment”, and “grim foreboding”,2 this literature demonstrates the need for research no only in



alternative methods of treatment, but also research into possible causative factors as well.

The remission of Patient E’s epilepsy is a phenomenon whose timing is shortly preceded by the

adjustment procedure. Perhaps not all seizure disorders can be helped with spinal adjustments. However,

chiropractic care would surely be the most conservative treatment in the primary care system. Careful

investigation into each case may reveal possible avenues that may eventually help the patient reduce or in

some cases entirely eliminate the need for drug therapy.

ACKNOWLEDGEMENTS

The authors wish to acknowledge George Hess, D.C., Chris L. Hendricks, D.C., Vern Hagen,

D.C., and Dale Strama, D.C. for their part in the care of Patient E and Alana Ferguson, M.S., Susan Larkin,

D.C. and David Guerriero for help in manuscript preparation.